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  1. Authors: Emily Vucic, Kelsie Thu, Victor Martinez, Chiara Pastrello, John Yee, Igor Jurisica, Stephen Lam and Wan Lam
    Citation: Cancer & Metabolism 2014 2(Suppl 1):P82

    This article is part of a Supplement: Volume 2 Supplement 1

  2. Authors: Julia Mayerle, Holger Kalthoff, Regina C Reszka, Beate Kamlage, Erik Peter, Bodo Schniewind, Sandra González-Maldonado, Volker Liebenberg, Christian Pilarsky, Philipp Schatz, Jonas A Schreiber, Ulrich F Weiss and Robert Grützmann
    Citation: Cancer & Metabolism 2014 2(Suppl 1):P60

    This article is part of a Supplement: Volume 2 Supplement 1

  3. Authors: Zachary Schug, Barrie Peck, Dylan Jones, Qifeng Zhang, Israt Alam, Tim Witney, Elizabeth Smethurst, Shaun Grosskurth, Adrian Harris, Susan Critchlow, Eric Aboagye, Michael Wakelam, Almut Schulze and Eyal Gottlieb
    Citation: Cancer & Metabolism 2014 2(Suppl 1):O9

    This article is part of a Supplement: Volume 2 Supplement 1

  4. The risk of pancreatic cancer, the 4th deadliest cancer for both men and women in the United States, is increased by obesity. Calorie restriction (CR) is a well-known dietary regimen that prevents or reverses ...

    Authors: Kristyn A Liu, Laura M Lashinger, Audrey J Rasmussen and Stephen D Hursting
    Citation: Cancer & Metabolism 2014 2:6
  5. Targeting cancer cell metabolism is recognized as a promising arena for development of cancer chemotherapeutics. Moreover, redox metabolism is also systematically altered in tumor cells. Indeed, there is growi...

    Authors: Shawn D Stuart, Alexandra Schauble, Sunita Gupta, Adam D Kennedy, Brian R Keppler, Paul M Bingham and Zuzana Zachar
    Citation: Cancer & Metabolism 2014 2:4
  6. Unlike glycolytic enzymes that directly catabolize glucose to pyruvate, the family of 6-phosphofructo-2-kinase/fructose-2,6-bisphosphatases (PFKFBs) control the conversion of fructose-6-phosphate to and from f...

    Authors: Alden C Klarer, Julie O’Neal, Yoannis Imbert-Fernandez, Amy Clem, Steve R Ellis, Jennifer Clark, Brian Clem, Jason Chesney and Sucheta Telang
    Citation: Cancer & Metabolism 2014 2:2
  7. Cancers cells shift their metabolism towards glycolysis in order to help them support the biosynthetic demands necessary to sustain cell proliferation and growth, adapt to stress and avoid excessive reactive o...

    Authors: Pearl Lee, Karen H Vousden and Eric C Cheung
    Citation: Cancer & Metabolism 2014 2:1
  8. A variety of oncogenic and environmental factors alter tumor metabolism to serve the distinct cellular biosynthetic and bioenergetic needs present during oncogenesis. Extracellular acidosis is a common microen...

    Authors: Gregory LaMonte, Xiaohu Tang, Julia Ling-Yu Chen, Jianli Wu, Chien-Kuang Cornelia Ding, Melissa M Keenan, Carolyn Sangokoya, Hsiu-Ni Kung, Olga Ilkayeva, László G Boros, Christopher B Newgard and Jen-Tsan Chi
    Citation: Cancer & Metabolism 2013 1:23
  9. Glutamine metabolism is a central metabolic pathway in cancer. Recently, reductive carboxylation of glutamine for lipogenesis has been shown to constitute a key anabolic route in cancer cells. However, little ...

    Authors: Shawn McGuirk, Simon-Pierre Gravel, Geneviève Deblois, David J Papadopoli, Brandon Faubert, André Wegner, Karsten Hiller, Daina Avizonis, Uri David Akavia, Russell G Jones, Vincent Giguère and Julie St-Pierre
    Citation: Cancer & Metabolism 2013 1:22
  10. Endocrine FGF21 and FGF19 target adipocytes and hepatocytes through betaKlotho (KLB) and FGFR tyrosine kinases effecting glucose, lipid and energy metabolism. Both factors alleviate obesity and metabolic abnor...

    Authors: Yongde Luo, Chaofeng Yang, Min Ye, Chengliu Jin, James L Abbruzzese, Mong-Hong Lee, Sai-Ching J Yeung and Wallace L McKeehan
    Citation: Cancer & Metabolism 2013 1:21
  11. Although cells require nutrients to proliferate, most nutrient exchange rates of the NCI60 panel of cancer cell lines correlate poorly with their proliferation rate. Here, we provide evidence indicating that t...

    Authors: Sonia C Dolfi, Leo Li-Ying Chan, Jean Qiu, Philip M Tedeschi, Joseph R Bertino, Kim M Hirshfield, Zoltán N Oltvai and Alexei Vazquez
    Citation: Cancer & Metabolism 2013 1:20
  12. Most normal cells in the presence of oxygen utilize glucose for mitochondrial oxidative phosphorylation. In contrast, many cancer cells rapidly convert glucose to lactate in the cytosol, a process termed aerob...

    Authors: Julia Billiard, Jennifer B Dennison, Jacques Briand, Roland S Annan, Deping Chai, Mariela Colón, Christopher S Dodson, Seth A Gilbert, Joel Greshock, Junping Jing, Hong Lu, Jeanelle E McSurdy-Freed, Lisa A Orband-Miller, Gordon B Mills, Chad J Quinn, Jessica L Schneck…
    Citation: Cancer & Metabolism 2013 1:19
  13. Germline and somatic mutations in STK11, the gene encoding the serine/threonine kinase LKB1, are strongly associated with tumorigenesis. While loss of LKB1 expression has been linked to breast cancer, the mechani...

    Authors: Fanny Dupuy, Takla Griss, Julianna Blagih, Gäelle Bridon, Daina Avizonis, Chen Ling, Zhifeng Dong, Doris R Siwak, Matthew G Annis, Gordon B Mills, William J Muller, Peter M Siegel and Russell G Jones
    Citation: Cancer & Metabolism 2013 1:18
  14. The Ets-1 proto-oncogene is frequently upregulated in cancer cells, with known involvement in cancer angiogenesis, metastasis, and more recently energy metabolism. In this study we have performed various bioin...

    Authors: Meghan L Verschoor, Chris P Verschoor and Gurmit Singh
    Citation: Cancer & Metabolism 2013 1:17
  15. The resurgence of interest in cancer metabolism has linked alterations in the regulation and exploitation of metabolic pathways with an anabolic phenotype that increases biomass production for the replication ...

    Authors: Jason A Zastre, Rebecca L Sweet, Bradley S Hanberry and Star Ye
    Citation: Cancer & Metabolism 2013 1:16

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