- Erratum
- Open Access
Erratum to: Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia
https://doi.org/10.1186/2049-3002-2-22
© Shukla et al.; licensee BioMed Central Ltd. 2014
- Received: 24 September 2014
- Accepted: 24 September 2014
- Published: 29 September 2014
The original article was published in Cancer & Metabolism 2014 2:18
Keywords
- Acetate
- Cancer Cell
- Cancer Research
- Quantitative Analysis
- Metabolic Disease
Correction
Ketone bodies inhibit growth and induce apoptosis in pancreatic cancer cell lines. Capan1 (A) and S2-013 (B) cells were treated with different concentrations of sodium-3-hydroxybutyrate (NaHB) and lithium acetoacetate (LiAcAc) for 72 h, and cell viability was determined by MTT assay. Bar represents percent viability under indicated treatments relative to treatment with solvent control. Representative bright-field images of Capan1 (C) and S2-013 (D) cells under treatment with 10- and 20-mM concentrations of NaHB and LiAcAc for 72 h. (E) Multiple pancreatic cancer cell lines were treated with 10- and 20-mM concentrations of NaHB and LiAcAc for 72 h, and relative cell viability determined by MTT assay is plotted in the bar charts. (F) Capan1 and S2-013 cells treated with 10- and 20-mM concentrations of sodium-3-hydroxybutyrate and lithium acetoacetate for 48 h and the relative caspase 3/7 activity are plotted. Values represented are mean ± SEM. *P<0.05; **P<0.01.
Notes
Declarations
Authors’ Affiliations
References
- Shukla SK, Gebregiworgis T, Purohit V, Chaika NV, Gunda V, Radhakrishnan P, Mehla K, Pipinos II, Powers R, Yu F, Singh PK: Metabolic reprogramming induced by ketone bodies diminishes pancreatic cancer cachexia. Canc Metabol. 2014, 2: 18-10.1186/2049-3002-2-18.View ArticleGoogle Scholar
Copyright
This article is published under license to BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/4.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly credited. The Creative Commons Public Domain Dedication waiver (http://creativecommons.org/publicdomain/zero/1.0/) applies to the data made available in this article, unless otherwise stated.