Fig. 6From: Prostate cancer cell proliferation is influenced by LDL-cholesterol availability and cholesteryl ester turnoverThe relationship between LDL-cholesterol metabolism, cholesteryl ester turnover, and proliferation of prostate cancer cell proliferation. The schematic shows that prostate cancer cell proliferation is reduced by pharmacological inhibition of a ACAT1 to block cholesterol esterification and reduced cholesteryl ester levels, or b neutral cholesteryl ester hydrolase (nCEH) activity which increased cholesteryl ester levelsBack to article page