Fig. 8From: HIF1/2-exerted control over glycolytic gene expression is not functionally relevant for glycolysis in human leukemic stem/progenitor cellsInhibition of BCR-ABL does impact on the glycolytic state of K562 cells, independent of HIF signaling and hypoxia. Glucose consumption (a) and lactate production (b) of K562 wt and ARNT knockout cells grown under normoxic and hypoxic growth conditions for 24 h cultured in the presence of increasing doses of imatinibBack to article page