Fig. 9From: A yeast phenomic model for the influence of Warburg metabolism on genetic buffering of doxorubicinWarburg-independent deletion enhancement of doxorubicin cytotoxicity. Gene interaction profiles showing deletion enhancement in both respiratory and glycolytic context included: a double-strand break repair via homologous recombination, and its child terms (indicated by arrows), and b the Cul8-RING ubiquitin ligase, Ino80 complex, Lst4-7 complex, and MCM complexBack to article page